Epidermal wound healing is a complex process that repairs injured tissue. The complexity
of this process increases when bacteria are present in a wound; the bacteria interaction
determines whether infection sets in. Because of underlying physiological problems
infected wounds do not follow the normal healing pattern. In this paper we present a
mathematical model of the healing of both infected and uninfected wounds. At the core of
our model is an account of the initiation of angiogenesis by macrophage-derived growth
factors. We express the model as a system of reaction-diffusion equations, and we present
results of computations for a version of the model with one spatial dimension.